What happens if aortic regurgitation occurs?

Aortic regurgitation is common in aortic valve insufficiency, which has two major types of factors: physiological and pathological. Many people have aortic insufficiency from birth. This is congenital, but if there are no symptoms, there is no need for treatment. There are also some pathological causes of aortic regurgitation that require everyone’s attention. The diseases that can cause aortic regurgitation are usually: rheumatic heart disease, valve disease, bacterial endocarditis and other factors. It is necessary to find the cause and symptomatic treatment. Regarding the question of what happens to aortic valve regurgitation, we must first understand what is the aortic valve and what is its working mechanism? The aortic valve is located between the left ventricle and the aorta, and its main function is to inhibit the blood flow injected into the aorta from flowing back into the left ventricle. If aortic valve insufficiency occurs, it will cause aortic blood reflux to the left ventricle. We often say aortic regurgitation. Aortic regurgitation will increase the burden on the left ventricle. Severe regurgitation can also cause heart failure and eventually lead to sudden death. Tips: In fact, mild aortic regurgitation does not have a great impact on the human body. There are many mild regurgitation conditions that do not require treatment. Of course, this needs to be diagnosed by a doctor before deciding whether or not to be treated. . For patients with moderate to severe aortic regurgitation, it is more dangerous, especially for patients who have already experienced symptoms of discomfort and need timely treatment.

What happens if aortic regurgitation occurs?

Aortic regurgitation is common in aortic valve insufficiency, which has two major types of factors: physiological and pathological. Many people have aortic insufficiency from birth. This is congenital, but if there are no symptoms, there is no need for treatment. There are also some pathological causes of aortic regurgitation that require everyone’s attention. The diseases that can cause aortic regurgitation are usually: rheumatic heart disease, valve disease, bacterial endocarditis and other factors. It is necessary to find the cause and symptomatic treatment. Regarding the question of what happens to aortic valve regurgitation, we must first understand what is the aortic valve and what is its working mechanism? The aortic valve is located between the left ventricle and the aorta, and its main function is to inhibit the blood flow injected into the aorta from flowing back into the left ventricle. If aortic valve insufficiency occurs, it will cause aortic blood reflux to the left ventricle. We often say aortic regurgitation. Aortic regurgitation will increase the burden on the left ventricle. Severe regurgitation can also cause heart failure and eventually lead to sudden death. Tips: In fact, mild aortic regurgitation does not have a great impact on the human body. There are many mild regurgitation conditions that do not require treatment. Of course, this needs to be diagnosed by a doctor before deciding whether or not to be treated. . For patients with moderate to severe aortic regurgitation, it is more dangerous, especially for patients who have already experienced symptoms of discomfort and need timely treatment.

Heart color Doppler ultrasound suggests mitral valve, what does mild tricuspid regurgitation mean? What should I do?

Many people will have this reminder after doing color Doppler ultrasound: mitral valve, tricuspid regurgitation, what does this mean? How should it be treated? 1. Mitral valve regurgitation 1. What is mitral valve. The heart has two atria and two ventricles. There is a door between the left atrium and the left ventricle. This door is the mitral valve. When the heart contracts, the heart goes out. When bleeding, this door is closed to prevent blood flow back to the left atrium. When the heart relaxes, this door opens, allowing blood from the left atrium to flow to the left ventricle. 2. Mitral valve regurgitation Mitral valve regurgitation is one of the common valve diseases. Due to congenital abnormalities or acquired diseases, the mitral valve cannot be completely closed when the left ventricle contracts, causing blood to flow from the left atrium into the left ventricle Part of the reflux returns to the left atrium, causing a series of pathological changes and clinical symptoms of the heart, which can lead to heart failure in severe cases. The structure of the mitral valve includes four parts: leaflets, annulus, chordae, and papillary muscles. The normal function of the mitral valve depends on the structural and functional integrity of these four parts and the left ventricle, and any one or more parts have structural abnormalities. Or dysfunction can cause mitral valve insufficiency. To put it simply, the door of the mitral valve, when closed, is not tightly closed, and a part of the blood will flow back to the left atrium. Over time, it may cause heart deformation and heart failure. 3. Morbidity The total prevalence of mitral regurgitation in adults over 35 years old in China is 18.4%, among which the prevalence of moderate to severe mitral regurgitation in the 35-50 years old population is 0.3%, 51-64 The prevalence rate for people aged 65 years is 0.9%, and that for people over 65 years old is 2.2%. The incidence trend increases with age. 4. Inspection methods Many years ago, most cardiovascular doctors could hear mitral valve problems through stethoscopes. However, with the popularity of ultrasound, it is now easy to detect mitral valve regurgitation through cardiac ultrasound, and can give A classification of mild to severe. Regurgitation area <4 square centimeters is mild; regurgitation area 4-8 square centimeters is moderate; regurgitation area> 8 square centimeters is severe; 2. What causes mitral regurgitation, simply speaking, some are congenital Sexual causes are partly caused by other heart diseases, and partly caused by age. For example, the direct cause of valve leaflet damage is rheumatic heart disease, which accounts for about 1/3 of mitral regurgitation. . Another example: mitral valve prolapse, congenital leaflet deformity, valve leaflet calcification, infective endocarditis, annulus enlargement, chordal rupture, papillary muscle ischemia, acute myocardial infarction and papillary muscle necrosis. With the increase of age, many people will have some mitral regurgitation due to the aging of the valve; Third, what is the manifestation of mitral regurgitation? Generally, mild chronic mitral regurgitation can have no manifestation, but if This kind of mitral regurgitation will continue to aggravate, and then there may be some manifestations of heart failure, such as dyspnea, fatigue, weakness, decreased activity endurance, edema of the lower limbs, and inability to lie down at night. If it is acute mitral regurgitation, it is more dangerous, and acute dyspnea may occur. If it is severe acute mitral regurgitation, there will be acute left ventricular manifestations, difficulty breathing, coughing pink foamy sputum, cardiogenic shock, and death. 4. What to do about mitral regurgitation? For those with no clear cause, such as mild or slow-progressive mitral valves in the elderly, no special treatment is required. It only takes six months to one year to review the color Doppler ultrasound observation. For those with a clear cause, such as rheumatism, endocarditis, and myocardial infarction, it appears to treat the primary disease. At the same time, those with heart failure should be treated according to the heart, and those with atrial fibrillation need anticoagulation to prevent thrombosis. In addition to these basic treatments, surgical treatment is also required for the more serious or rapid progress. Patients with severe mitral regurgitation and clinical symptoms; accompanied by pulmonary hypertension, etc. that require surgical treatment. Valve replacement is a kind of thoracotomy that removes the diseased mitral valve and replaces it with an artificial mechanical valve made of synthetic materials. Or a biological valve made of biological materials and fixed with special sutures. Interventional therapy can also be used, which is mainly suitable for patients who are not suitable for surgery, such as elderly patients, a history of thoracotomy, poor cardiac function and multiple organ dysfunction. Patients with severe acute mitral regurgitation caused by abnormalities of various papillary muscles, chordae and valve leaflets, if the operation is not performed in time, the mortality rate is extremely high; age >50 years, with enlarged left atrium and left ventricle , Poor prognosis

Heart mitral regurgitation, severe? How should it be treated? Cardiologist clearly

When many people go to the hospital to check the heart, they will be prompted after cardiac ultrasound: mitral valve insufficiency, or tricuspid valve insufficiency; or mitral valve stenosis, tricuspid valve stenosis, etc. results. So what do these mitral and tricuspid valves mean? 1. The mitral valve is a simple part of the heart. Our heart has two atria: the left atrium and the right atrium; the two ventricles: the left heart failure and the right ventricle; a door is needed between the left atrium and the left ventricle. The atrium is separated from the left ventricle, and this door is called the mitral valve. Between the right atrium and the right ventricle is the tricuspid valve. 2. What is the role of the mitral valve? The mitral valve is a “one-way valve” to ensure blood flow from the left atrium to the left ventricle unidirectionally. When the heart contracts, the blood in the left ventricle must be transported to the whole body. Let the blood of the left ventricle flow back to the left atrium. At this time, the mitral valve will be closed, blocking the passage of the left atrium and the left ventricle. When the heart contraction ends, that is, once the blood is delivered to the whole body, the left ventricle is empty. At this time, the heart relaxes, and the blood needs to be sent from the left atrium through the mitral valve into the left ventricle again. Open, successfully complete the left ventricular congestion, ready for the next heart contraction. This mitral valve is to prevent the blood from the left ventricle from flowing back to the left atrium. 3. Mitral valve insufficiency The normal function of mitral valve closure depends on the complete structure and normal function of the five parts of the valve leaflet, annulus, chordae, papillary muscle, and left ventricle. Any one of these five parts occurs Abnormal structure and function can cause mitral regurgitation. That is, when the mitral valve needs to be closed, the door cannot be completely closed, then the result is that when the heart is contracting, when the left ventricle supplies blood, part of the blood will return to the left atrium. Very mild mitral valve insufficiency, without any discomfort, can only be found when doing heart color Doppler ultrasound; Moderate mitral valve insufficiency, there may be some chest tightness, breathlessness, difficulty in breathing, etc.; poisoning mitral Valve insufficiency, especially acute mitral valve insufficiency, can be life-threatening. 4. The causes of mitral insufficiency There are many reasons for mitral insufficiency, which are roughly divided into chronic and acute onset. Chronic mitral valve insufficiency: 1. Rheumatic heart disease: after rheumatic fever, it can cause rheumatic heart disease, the mitral valve can not be closed gradually, the mitral valve insufficiency caused by rheumatic heart accounts for all mitral One third of patients with insufficiency of flaps are more common in men. 2. Congenital heart disease: dysplasia of heart during fetal period, abnormal mitral valve development, congenital malformation, and mitral valve insufficiency at birth. It is most commonly seen in endocardial cushion defects or corrected heart transposition; endocardial elastin fibrosis; parachute mitral valve deformity. 3. Degeneration in the elderly: As the age increases, the mitral valve will gradually age, and patients with severe aging will not normally close due to calcification, which is more common in elderly women. 4. Enlargement of the left ventricle When the left ventricle is enlarged, that is, the house is enlarged, the door is not changed, and naturally the door is not fully closed. 5. Other diseases There are also some diseases such as connective tissue diseases such as systemic lupus erythematosus, rheumatoid arthritis, etc.; hypertrophic obstructive cardiomyopathy; ankylosing spondylitis and other diseases may cause incomplete mitral regurgitation. For patients with chronic mitral insufficiency, mild asymptomatic, can continue to observe, without medical treatment, medical treatment can not change mitral insufficiency. For those who have already developed symptoms or combined with heart failure, they need to be treated according to heart failure, heart failure, etc. to prevent the increase of heart failure. Acute mitral valve insufficiency Acute myocardial infarction often has some complications, such as rupture of tendon cord, necrosis of papillary muscle, etc., which will lead to acute mitral valve insufficiency, then it will lead to acute cardiac insufficiency, leading to acute left heart Fading, life-threatening. At this time, comprehensive rescue treatment is needed. For the mitral valve insufficiency that continues to worsen, surgical treatment is required to fundamentally resolve the mitral valve insufficiency. In short, when we finish the color Doppler ultrasound and find mitral regurgitation, we must first find the cause; and decide the next treatment plan, or observation, or medicine, or surgery according to the degree of mitral regurgitation. However, for the slow progress, mitral valve insufficiency that does not cause complications, and no need to be over-stressed, it is enough to regularly review the color Doppler ultrasound.

21 year old college student suffering from hypertension and heart disease

The senior students suffered a lot of pressure because of the job search, and they often felt dizzy. They even got high blood pressure during the investigation, and they were also accompanied by enlarged left ventricle (ie, hypertension and heart disease). Experts said yesterday that high blood pressure is no longer a patent for the elderly, and that young people are suffering from more and more hypertension, mainly related to irregular diet, long-term stay up late, mental stress and excessive stress. 21-year-old Chen Fei is a senior student and is about to graduate, but his work has not yet finished. From the second semester of his junior year on, his job search has not been smooth. Several written tests have been passed, and he was brushed down due to excessive tension during the interview. Because of poor family conditions, he was under a lot of psychological pressure. He has been having poor sleep at night for more than half a year and often feels dizzy. In recent days, Chen Fei not only had flowers in his eyes, but also felt dizzy while lying in bed. Upon examination at the hospital, the doctor found that his blood pressure was as high as 160/100 mm Hg. The 24-hour ambulatory blood pressure monitoring results were also unsatisfactory, and he had left ventricular hypertrophy, which was eventually diagnosed as hypertensive heart disease. Knowing this result, Chen Fei couldn’t believe it, how could he get high blood pressure when he was only 21 years old? After a systematic examination, the doctor ruled out secondary hypertension and was diagnosed with primary hypertension. The doctor believes that he may be hypertension caused by long-term mental stress and stress. “Young people are prone to vasoconstriction and increased blood pressure because of increased work pressure and mental stress, and they can easily develop hypertension in the long run.” Yang Feiyan, deputy chief physician of the Department of Cardiology of the hospital, said that increased blood pressure will cause heart disease Damage, when the blood pressure rises, the burden on the left ventricle becomes heavier, and the left ventricle is affected for a long time, and it will gradually become thickened and enlarged due to compensation, which is hypertension and heart disease. It is understood that high blood pressure has always been considered as a geriatric disease in recent years, and there has been a trend of youth in recent years. There are also many young people in the 20s and 30s who suffer from essential hypertension, mainly related to poor living habits, such as staying up late , Less exercise, obesity, smoking and drinking. At the same time, white-collar workers who are in a state of intense social competition and work pressure, and who have been in a state of tension for a long time are also high-risk people with hypertension. Remind that high blood pressure will not cause discomfort in the early stage, so many patients with high blood pressure do not effectively control blood pressure, and eventually complications such as myocardial infarction, stroke, and renal failure occur. Adults are advised to measure their blood pressure at least once a year, and those with a family history every three months. In addition, it is necessary to develop healthy lifestyle habits and learn to adjust the mentality and relieve stress when the mental stress is high. Once diagnosed with high blood pressure, medication should be given under the guidance of a doctor.

What is the cause of aortic stenosis?

 What is aortic stenosis? Is it seriously life-threatening if I have the disease? In fact, this disease is a type of heart disease. This disease is caused by congenital or acquired factors that cause aortic valve lesions that cause the aortic valve to not fully open during systole.  Aortic valve stenosis can be caused by a variety of reasons. The causes of aortic valve stenosis are mainly caused by the sequelae of rheumatic fever, congenital aortic valve structural abnormalities or senile aortic valve calcification. Congenital malformations are often caused by an abnormal number of leaflets, and the two-leaf type is the most common. Rheumatic valvular disease usually has both stenosis and insufficiency. Acquired non-rheumatic valve disease is mainly degenerative aortic valve sclerosis in the elderly, often accompanied by extensive atherosclerosis or hypercholesterolemia. The disease often develops slowly and has a long asymptomatic period, but the prognosis is poor, and the condition will deteriorate rapidly after heart failure. Aortic valve stenosis is caused by chronic recurrent rheumatic fever that causes swelling of the aortic valve annulus, fusion adhesion at the junction, contraction and stiffness of the free edge of the valve tip, and calcified nodules can be produced on the surface of the valve leaflets, causing the valve orifice narrow. These pathological changes often cause stenosis and reflux to coexist. Rheumatic aortic valve disease is often accompanied by mitral valve disease. ① In the decompensated period, due to the increased left ventricular systolic pressure, the left ventricular weight index increases and the myocardial oxygen consumption is at a high level, so obvious aortic valve stenosis can produce increased myocardial oxygen demand on the one hand, and on the other hand, due to the coronary The relative decrease in pulse flow leads to insufficient oxygen supply, which leads to ischemia of the subendocardial myocardium. When myocardial contractility is decompensated and weakened, left heart function declines. Finally, as myocardial degeneration decreases, the left ventricle dilates and the ejection fraction decreases. As a result, the left ventricular end-diastolic pressure further increased leading to pulmonary venous hypertension, resulting in chronic pulmonary congestion. Aortic valve stenosis increases myocardial oxygen demand and subendocardial perfusion pressure, which can cause arrhythmia, chest pain, and even sudden cardiac death. Adults can also be accompanied by coronary heart disease and aggravate myocardial ischemia. Long-term pulmonary congestion can gradually produce pulmonary hypertension, resulting in right ventricular hypertrophy and dilation, which ultimately leads to right heart failure. ②The left atrial ventricular compensation period is about 3.0cm2, and when the orifice area is reduced to 1.0cm2, the left ventricular blood flow is blocked and the systole is overloaded, resulting in a larger space between the left ventricle and the aorta Systolic pressure step difference, normal pressure step difference is 0.67 kPa (5 mm Hg), mild stenosis is 0.67 to 2.67 kPa (5 to 20 mm Hg), moderate stenosis is 2.67 to 6.67 kPa ( 20 to 50 mm Hg), more than 6.67 kappa (50 mm Hg) in severe stenosis (equivalent to the valve area reduced to less than a quarter of the normal, that is, the valve opening is less than 0.8 cm2). As the aortic valve stenosis worsens, the left ventricular concentric hypertrophy compensates for the increased pressure in the left ventricular cavity.  About the cause of aortic valve stenosis, we have learned through a brief introduction of the above related knowledge. At the same time, after learning the relevant disease knowledge, we must pay attention to our body in daily life, try not to be entangled by the terrible disease, affecting normal life.

Can a newborn baby with high blood pressure and aortic stenosis be cured?

&nbsp.&nbsp.&nbsp.&nbsp. In people’s consciousness, high blood pressure is a disease that only exists in adults, and it has nothing to do with children. However, a few days ago, in the neonatal department of Changzhou Maternal and Child Health Hospital, in the process of rescuing a premature baby, it was found that this child had symptoms of high blood pressure, which was puzzled. What is going on? ? At that time, a mother gave birth to twins, and the hypertensive baby was the smaller one of the premature twins, with a birth weight of only 1.3 kg. But the blood pressure is almost 100, the systolic blood pressure is 100, and the diastolic blood pressure is 70. &nbsp.&nbsp. What is even more strange is that there is a clear difference between the blood pressure of the upper extremity and the lower extremity of the newborn. A dozen or so, such blood pressure is very risky for premature babies of a few kilograms and can be dangerous at any time. &nbsp.&nbsp.&nbsp.&nbsp. After a case discussion, the doctor highly suspected that it was caused by aortic stenosis, contacted the radiology department, and gave the baby enhanced ct, that is, after angiography, it was found that the stenosis in the aorta was only 1.5 mm, very narrow, the front section is 5 mm, the back section is 7 mm, and the middle is 1.5 mm, so it can’t pass the blood to this place, and the blood pressure is relatively high. At present, the baby has been transferred to the Children’s Hospital for follow-up treatment. &nbsp.&nbsp.&nbsp.&nbsp. When it comes to aortic stenosis, many people are not aware of this disease. The mechanism of the occurrence of aortic stenosis in the fetus is more complicated. It is a kind of abnormal anatomical structure caused by the development of the heart and large blood vessels of the fetus for some reason in the first trimester. It is a kind of congenital disease of the fetus. The incidence is relatively low. We know that the aorta and the left ventricle are connected. If the aorta is significantly narrowed, the blood flow of the left ventricle is blocked and the left ventricle is significantly burdened. After a long time, the left ventricle is obviously hypertrophic, which will damage the heart function, affect the child’s quality of life, and even threaten the child’s life. &nbsp.&nbsp.&nbsp.&nbsp. Against this congenital disease of the fetus, pregnant mothers should not worry too much. Generally, four-dimensional ultrasound is performed at 18-20 weeks of pregnancy to check the fetal heart structure, and abnormalities of the fetal heart structure can be found in time. If fetal aortic stenosis is found, it is best to ask a doctor specializing in cardiac surgery to evaluate the condition. If the aortic stenosis is not serious, you can choose surgical treatment after birth. The success rate is relatively high, about 90%. The sooner the timing of the operation, the better (3-6 months). The operation time should be advanced for those with severe symptoms. &nbsp.&nbsp.&nbsp.&nbsp. In our hospital, some women have neonatal aortic stenosis, but the symptoms are not too serious, so they ask, is it conservative treatment? Some people even feel that slowly, as the child develops, they will grow up without treatment. This idea is wrong. Neonatal aortic stenosis has a great influence on hemodynamics, which will seriously affect the growth and development of children. Generally, they cannot grow up on their own, and surgery needs to be performed as soon as possible. &nbsp.&nbsp.&nbsp.&nbsp. Parents are better to take their children to the heart surgery examination of the top three hospitals, and treat them under the guidance of doctors. If the condition is mild, you can continue to observe. If you need surgical treatment, you can choose surgery according to the condition and the baby’s physical condition. Before surgery, pay attention to scientific feeding to avoid children catching colds. If the aortic stenosis is detected in the fetal stage, the condition is serious, and it has a great impact on health after birth. The pregnancy may need to be terminated. The pregnant woman needs to be mentally prepared. Having a healthy baby is more important than anything. &nbsp.&nbsp.&nbsp.&nbsp.

Symptoms of aortic insufficiency

The onset of many heart diseases will not only affect the lives of patients, but also worry and fear the relatives of patients. Therefore, in the face of the torture of heart disease, we must encourage and support patients to receive scientific treatment, and we must also understand more about Medical knowledge about patients’ diseases can better help patients fight against diseases. · What is aortic valve insufficiency? This disease refers to the destruction of any one of the aortic valve annulus, aortic sinus, aortic valve leaflet, valve junction, and aortic sinus canal junction, resulting in poor closure of the aortic valve leaflet during diastole. The main influencing factors of late outcome after aortic valve insufficiency are still left ventricular cavity size and left ventricular function. Therefore, to attract the attention of patients and doctors, prognostic care conditioning is also very important. · Understanding the symptoms of aortic valve insufficiency Aortic valve insufficiency can be caused by aortic valve and annulus, as well as ascending aortic lesions. At present, male patients with the disease are more common, accounting for about 75%; but female patients with the disease are often accompanied by mitral valve disease. Among the chronically ill patients, the damage caused by rheumatic fever is the most common, which accounts for 2/3 of all patients with aortic insufficiency. Regarding the symptoms of aortic valve insufficiency, usually, patients with aortic valve insufficiency are asymptomatic for a long period of time, even if the obvious aortic valve insufficiency can be more than ten years until the obvious symptoms appear, once the heart power Failure, progress quickly. Therefore, we must keep in mind the following clinical symptoms of the disease to be alert to the sudden onset of the disease: ① syncope: When changing position rapidly, dizziness or dizziness may occur, and syncope is rare. ② Chest pain: Angina is less common than aortic valve stenosis. Chest pain may be caused by excessive stretching of the ascending aorta or significant enlargement of the heart during left ventricular ejection. There are also factors of myocardial ischemia. Occurs at rest and has a long duration, and has a poor response to nitroglycerin. The onset of nocturnal angina pectoris may be due to a further decrease in diastolic blood pressure due to a slower heart rate at rest, which reduces coronary blood flow. Those with abdominal pain may be related to visceral ischemia. ③ dyspnea: exertional dyspnea first appeared, indicating that the heart reserve capacity has been reduced, as the disease progresses, there may be end-to-end breathing and paroxysmal dyspnoea at night. ④ Palpitations: The discomfort of the heart beat may be the earliest complaint, due to the obvious enlargement of the left ventricle and the increase in apical beats, especially in the left or prone position. Emotional or physical activity causes tachycardia, or Ventricular premature beats can make palpitation more obvious. Due to the significant increase in pulse pressure, there are often strong arterial pulses in all parts of the body, especially the head and neck. ⑤Acute aortic valve insufficiency, due to the sudden increase in left ventricular volume load, increased wall tension, left ventricular expansion, acute left heart failure or pulmonary edema can occur quickly. ⑥Other symptoms: fatigue, significant decline in activity endurance, excessive sweating, especially when there is paroxysmal dyspnoea at night or angina pectoris at night, hemoptysis and embolism are rare, and liver congestion and swelling can occur in late right heart failure. There is tenderness, edema of the ankle, pleural effusion or ascites. The sudden death of many heart disease patients is not without cause. While we pay attention to the disease itself, we must also pay attention to the patient’s emotions and pressures, especially for heart disease patients. It is common sense not to be stimulated, so we work as family members and medical services People must deal with patients’ negative emotions and psychological stress.

What is the cause of aortic stenosis?

What is aortic stenosis? Is it seriously life-threatening if I have the disease? In fact, this disease is a type of heart disease. This disease is caused by congenital or acquired factors that cause aortic valve lesions that cause the aortic valve to not fully open during systole. According to medical research, the aortic valve stenosis can be caused by many reasons. The causes of aortic valve stenosis are mainly caused by the sequelae of rheumatic fever, congenital aortic valve structural abnormalities or senile aortic valve calcification. Congenital malformations are often caused by an abnormal number of leaflets, and the two-leaf type is the most common. Rheumatic valvular disease usually has both stenosis and insufficiency. Acquired non-rheumatic valve disease is mainly degenerative aortic valve sclerosis in the elderly, often accompanied by extensive atherosclerosis or hypercholesterolemia. The disease often develops slowly and has a long asymptomatic period, but the prognosis is poor, and the condition will deteriorate rapidly after heart failure. Aortic valve stenosis is caused by chronic recurrent rheumatic fever that causes swelling of the aortic valve annulus, fusion adhesion at the junction, contraction and stiffness of the free edge of the valve tip, and calcified nodules can be produced on the surface of the valve leaflets, causing the valve orifice narrow. These pathological changes often cause stenosis and reflux to coexist. Rheumatic aortic valve disease is often accompanied by mitral valve disease. ① In the decompensated period, due to the increased left ventricular systolic pressure, the left ventricular weight index increases and the myocardial oxygen consumption is at a high level, so obvious aortic valve stenosis can produce increased myocardial oxygen demand on the one hand, and on the other The relative decrease in pulse flow leads to insufficient oxygen supply, which leads to ischemia of the subendocardial myocardium. When myocardial contractility is decompensated and weakened, left heart function declines. Finally, as myocardial degeneration decreases, the left ventricle dilates and the ejection fraction decreases. As a result, the left ventricular end-diastolic pressure further increased leading to pulmonary venous hypertension, resulting in chronic pulmonary congestion. Aortic valve stenosis increases myocardial oxygen demand and subendocardial perfusion pressure, which can cause arrhythmia, chest pain, and even sudden cardiac death. Adults can also be accompanied by coronary heart disease and aggravate myocardial ischemia. Long-term pulmonary congestion can gradually produce pulmonary hypertension, resulting in right ventricular hypertrophy and dilation, which ultimately leads to right heart failure. ②The area of ​​the normal aortic valve in the left ventricular compensation stage is about 3.0cm2. When the valve area is reduced to 1.0cm2, the left ventricular blood flow is blocked and the systolic load is excessive, resulting in a greater contraction between the left ventricle and the aorta Stage pressure gradient, normal pressure gradient 0.67 kPa (5 mm Hg), mild stenosis 0.67 to 2.67 kPa (5 to 20 mm Hg), moderate stenosis 2.67 to 6.67 kPa (20 To 50 mm Hg), more than 6.67 kappa (50 mm Hg) in severe stenosis (equivalent to the valve area reduced to less than a quarter of the normal, that is, the valve opening is less than 0.8 cm2). As the aortic valve stenosis worsens, the left ventricular concentric hypertrophy compensates for the increased pressure in the left ventricular cavity. About the cause of aortic valve stenosis, we have learned through a brief introduction of the above related knowledge. At the same time, after learning the relevant disease knowledge, we must pay more attention to our bodies in daily life, try not to be entangled by terrible diseases, and affect normal life.

Buying health care products spends money and is counted by the daughter-in-law, resulting in a heart like an octopus! Finally stop jumping!

Anger and anger will not only cause heart disease, but even cause heart disease and death! A patient was admitted a year ago, Aunt Lai. Aunt Lai Lai ’s son “secretly” gave 3,000 yuan to her mother, saying that it was almost New Year and buying something. Later, Aunt Lai, like many elderly people, usually likes to listen to health care. Auntie bought all of the health care products with 3000. After returning home, the family found that the health care products were not as good as propaganda. This matter was also known to the daughter-in-law, who not only scolded her son, but also counted many unpleasant words about her mother-in-law. My aunt felt unreasonable. She was sullen in her life. She didn’t eat or drink. During supper, the aunt suddenly had chest pain and sweaty. 120 arrived. After finishing the electrocardiogram, it was a heart attack! Blood pressure 80/40, has been shocked. Our angiography found that the three blood vessels did not stenosis at all; learning what happened during the day, we did a left ventricular angiography and found that the apical balloon-like change was originally a heart disease that was out of breath, called stress cardiomyopathy. Later, he was sent back to the intensive care unit, and his condition became more and more serious at night. Finally, he failed to rescue him. 1. Stress cardiomyopathy is a heart disease caused by anger and depression. This kind of heart disease often occurs due to sudden emotional excitement, startle, quarrel, excitement, fear, sadness and other predisposing factors. 80% of the disease occurs in menopausal women over 60 years old, and the incidence rate is 6-9 times that of men. . The onset is almost the same as acute myocardial infarction, which occurs more than after stress. It is characterized by sudden angina-like symptoms, transient left ventricular systolic dysfunction, ST-T changes in the electrocardiogram, left ventricular angiography or echocardiography , Left ventricular apical ventricular dyskinesia and apical balloon-like changes, myocardial enzyme spectrum can be slightly increased, often misdiagnosed as acute myocardial infarction, but coronary angiography often has no obvious stenosis. Although most of the prognosis is better than myocardial infarction, a small number of people still have complications such as heart failure, arrhythmia, and even death. This disease is often manifested by a sudden thinning of a myocardium in the left ventricle, which is swollen like a balloon by the impact of blood and resembles the head of an octopus, so it is called myocardial balloon-like change or octopus crown heart disease, and because of this The cause of this disease is usually related to emotions, so it is also called stress heart disease. This disease was first discovered in Japan, and was later recognized by Europe, America and even the world. Aunt Lai was angry because of this heart disease, and ultimately failed to rescue. Being angry can not only cause this kind of stress heart disease. Three years ago, we treated a patient with acute myocardial infarction. The old man was usually grumpy. He did n’t listen to anyone. After finishing the stent, the blood vessel was opened. The patient himself was not uncomfortable, and the old man was discharged from the hospital, no one said it, and beat him; the family could not control it; the acute left heart failure occurred in the alarm and eventually died. 2. Study an article in “Circulation” to prove that extreme anger is not a good thing for the patient or for others. Strong emotions or actions can cause sympathetic nerve excitement and increase the body’s catecholamine levels, which can lead to increased blood pressure and heart rate, vasoconstriction, and may even cause plaque rupture of blocked arteries, impaired coronary blood supply, and thus promote heart Disease attack. Anger is closely related to heart disease. We can often hear who gets mad with myocardial infarction and who gets mad with heart disease. This not only stays in the folk saying, research has confirmed it! The study also confirmed that extreme emotions such as anger and anxiety increased the risk of heart attack by 3 to 8.5 times within two hours. Anxiety is more dangerous and increases the risk of heart attack by 9.5 times. Anger can increase our risk of heart disease by 750% within 2 hours; 3. Clinically, I have worked in the Department of Cardiology for 20 years. The patients I met have not only relied on the aunt, but also the elderly with heart failure after myocardial infarction; I have been to many cardiomyopathy, myocardial infarction, heart failure, arrhythmia and other diseases caused by bad emotions, or anger, or anger, or depression, or anxiety and other negative emotions. If we can’t understand this matter, we can do a small experiment. When you are in a bad mood and the negative emotions are serious, you can measure your blood pressure. At this time, your blood pressure is obviously increased, and your heart rate is obviously accelerated; and when you are in a happy mood , Happy, happy, optimistic, when measuring blood pressure at this time, it must be more stable and a slower heart rate. Heart rate and blood pressure