Parkinson’s disease, also known as tremor paralysis, is one of the most common neurodegenerative diseases. The etiology and pathogenesis are not yet clear, and may be related to social factors, drug factors, patient factors, etc. Sexual dysfunction encountered by patients with Parkinson’s disease is often a problem that is ignored by patients and doctors for various reasons. However, the current study found that the rate of sexual dysfunction in patients with Parkinson’s disease is significantly higher than that of the same age , The normal population of the sex group, and other people with chronic diseases of the non-neurological system, especially the married male patients are the most significant, usually manifested as coldness, impotence, premature ejaculation and other symptoms, resulting in low sexual life satisfaction. Elderly male patients usually suffer from high levels of sexual dysfunction due to their own functional decline, even without Parkinson’s disease. Because Parkinson’s disease mainly affects the elderly over 55 years old, there has been little research on the sexual function of young and middle-aged patients (35-55 years old). Recent studies have found that this group of patients also has high incidence of sexual dysfunction, and it is particularly obvious in young women. It may be closely related to female psychological factors and the impact on menstrual cycle. Some patients’ sexual partners (husbands/wifes) will also be affected by One side of the disease affects sexual dysfunction to varying degrees. Parkinson’s patients may wish to do rehabilitation exercises. Experts say that Parkinson’s patients suffer from involuntary limb shaking and slow movements, and suffer great physical and mental pain. It is recommended that patients can use some sports to perform rehabilitation exercises based on adherence to standardized treatment, and help them improve their quality of life. The following three rehabilitation exercises can enable Parkinson’s disease patients to perform at home-turn around and lie down, hands are naturally placed on the side of the body, both legs are bent hips and knees, and feet are standing on the bed. Cross your fingers with your fingers and swing left and right, vigorously swing to one side, turn your head to the same side, swing your knees to the same side, try to make the hands and knees of the same side touch the bed. Sit in a soft and moderate bed. Keep your knees as straight as possible. Keep your feet as close as possible to the bed backboard or wall. Stretch your hands forward and touch your lower toes. If the patient has difficulty bending down, they can be assisted by a family member. The family member stands behind the patient, hands are placed on the patient’s back, and gently press forward slowly. By practicing this action, you can make Pa You walk more stable and not easy to fall. 、 Standing position, feet slightly apart, hands naturally placed on the side of the body. Stepping, coordinated swinging of both hands, walking in a straight line, the step size and step width gradually decrease from large, so that the two feet walk as small as possible in a straight line. Pay attention to safety during walking to prevent falls. It’s more convenient for Pa You to practice this action at home. The training principle is to go step by step. At the beginning, I was afraid of instability. The legs can be separated and leaned on a straight line. line.
Jumping eyelids, always twitching his face, is it sick? Need treatment? Such patients are often encountered in outpatient clinics. The eyelids jump on one side, and the face always twitches, which is medically called facial muscle spasm and is strictly a disease. Similar to the pathogenesis of trigeminal neuralgia, the onset of facial spasm is also related to nerve compression by blood vessels. Recurrent seizures of facial muscle spasms are paroxysmal, involuntary twitching, which is exacerbated when the emotion is agitated or tense, and in severe cases may cause difficulty in opening eyes, skewed corners of the mouth, and twitch-like murmurs in the ear. Because the initial symptom of facial muscle spasm is eyelid beating, the people are also known as “left eye jump wealth, right eye jump disaster”, so it generally does not attract people’s attention, and after a period of time, it develops into facial muscle spasm, which moves to the corner of the mouth , Severe joint neck. Facial muscle spasm can be divided into two types, one is primary facial muscle spasm, and the other is secondary facial muscle spasm, that is, facial muscle spasm caused by facial paralysis sequelae. Two types can be distinguished from the symptoms. Facial spasm of the original hair type can also occur at rest. Spasm relieves after a few minutes and is not controlled. Facial spasm caused by the sequelae of facial paralysis is only caused by blinking and eyebrow raising. Clinical manifestations of facial spasm Most primary facial spasm develops after middle age, and more women. The initial stage of the disease is mostly paroxysmal involuntary twitching of the orbicularis oculi muscle on one side, which gradually slowly expands to the other facial muscles on one side of the face. The twitching of the oral muscles is the most noticeable. , But the frontal muscles are less involved. The degree of twitching varies from paroxysmal, rapid and irregular. At first, the convulsions were lighter and lasted only a few seconds. Afterwards, the twitch gradually increased to a few minutes or longer, while the intermittent time was gradually shortened, and the convulsions gradually became more frequent. In severe cases, it is ankylosing, so that the ipsilateral eyes cannot be opened, the corners of the mouth are skewed to the same side, and they are unable to speak. They are often exacerbated by fatigue, nervousness, and voluntary movement, but they cannot imitate or control their attacks on their own. A convulsion can be as short as a few seconds and last for more than ten minutes. The duration of the intermittent period is uncertain. The patient feels upset and unable to work or study, which seriously affects the physical and mental health of the patient. Most convulsions stop after going to sleep. Bilateral muscle spasms are rare. If there is, it is usually on both sides. After one side of the convulsions stops, the other side relapses, and the convulsions are mild on one side and the other side is serious. Both sides have simultaneous onset and no convulsions have been reported. A small number of patients have mild facial pain during convulsions, and individual cases may be accompanied by ipsilateral headache and tinnitus. How to confirm the diagnosis of facial spasm? The clinical diagnosis of facial muscle spasm is mainly based on the typical symptoms of the patient: involuntary, paroxysmal, recurrent seizures of the unilateral muscles, mental tension, anxiety and excessive psychological pressure will induce or aggravate the seizures, and facial muscles such as exertion of force Closing eyes and bulging cheeks can also induce seizures, even during sleep and anesthesia. For patients lacking characteristic clinical manifestations, they need to be clarified with the help of auxiliary examinations, including electrophysiological examinations, imaging examinations, and carbamazepine treatment trials.
& nbsp. & nbsp. Facial muscle spasm, also known as facial muscle twitching, is more common in middle-aged and older ages. The onset usually starts from the orbicularis oculi muscle on one side, which is manifested by eyelid beating, paroxysmal, involuntary convulsions, and gradually expands to the other facial muscles on one side of the face. The convulsions of the mouth and corner muscles are the most noticeable. Involving the latissimus dorsi muscle on the same side, but the frontal muscles are less involved. & nbsp. & nbsp. The degree of convulsions varies from paroxysmal, rapid and irregular convulsions. At first, the convulsions were lighter and lasted only a few seconds. Afterwards, the twitch gradually increased to a few minutes or longer, while the intermittent time was gradually shortened, and the convulsions gradually became more frequent. In severe cases, it is ankylosing, so that the ipsilateral eyes cannot be opened, and the corners of the mouth are skewed to the same side, unable to speak, often exacerbated by fatigue, nervousness, and voluntary movement, but they cannot control their seizures by themselves. & nbsp. & nbsp. The patient feels upset and unable to work or study, which seriously affects the physical and mental health of the patient. Most convulsions stop after going to sleep. Bilateral muscle spasms are rare. A small number of patients have mild facial pain during convulsions, and individual cases may be accompanied by ipsilateral headache and tinnitus. & nbsp. & nbsp. The best drugs for the treatment of facial muscle spasm are carbamazepine and oxcarbazepine. There are many treatment methods for facial muscle spasm, including Chinese medicine, western medicine, acupuncture, closed needle, and surgery. The conventional treatment is medicine, mainly Western medicine, and the best medicines are also commonly used are carbamazepine and oxcarbazepine. The doctor will give an appropriate dose according to the patient’s condition. After taking carbamazepine or oxcarbazepine in patients with hemifacial spasm initially, the effect was significant, the convulsions disappeared, and a small number of patients could not tolerate side effects. If you can’t tolerate the side effects of the drug, go to the hospital in time. & nbsp. & nbsp. Western medicine is the main, supplemented by traditional Chinese medicine, acupuncture and closed needle treatment are prone to recurrence, and should not be used too much to avoid sequelae such as facial paralysis. There are also many surgical procedures to treat facial muscle spasm. It cannot be chosen blindly. Patients need to understand their condition, whether it is primary facial muscle spasm or secondary facial muscle spasm. & nbsp. & nbsp. The primary facial muscle spasm has no clear etiology. It is now generally believed that the facial auditory nerve is compressed by tortuous intracranial blood vessels. According to this principle, microvascular decompression treatment was carried out. Microsurgical techniques were used during the operation. , Observe and find the responsible blood vessels and nerves, carefully separate the two, and place decompression materials between the two, so that the convulsions disappear.