For more patient communication and help, please pay attention to the WeChat public account [Cardiology Patients Association] Acute myocardial infarction is on the cusp. Many people may have heard that acute myocardial infarction requires cardiac stent treatment, but this does not guarantee that the patient will be rescued. The cost is also relatively high, which may be a big burden for some families. Therefore, some people hope to treat it in a conservative way. Can acute myocardial infarction be treated conservatively after all? The most important part of the treatment of acute myocardial infarction is to open the infarct-related blood vessel. Two major methods are usually used for the infarction-related blood vessel. One is PCI, which is stent implantation, and the other is thrombolysis. The best time for thrombolysis is within three hours of the onset of symptoms, but when most patients go to the hospital for a clear diagnosis of acute myocardial infarction, the symptoms have lasted more than three hours, so PCI intervention is needed to open the relevant blood vessels of the infarction to save Ischemic or dying myocardium. The effect of conservative treatment with drugs alone is relatively limited. Even if conservative treatment with drugs including intravenous thrombolytic therapy is performed, patients need to undergo coronary angiography within a short period of time to understand the condition of coronary artery disease. Conservative treatment with drugs alone cannot determine the coronary artery condition. Patients may Myocardial infarction occurred again in a short time. Through the above introduction, everyone has also learned that the best treatment for acute myocardial infarction is stent therapy, but conservative treatment can also be adopted for patients who are really in difficulty. After all, family members also need to live on. I hope everyone can maintain a good attitude and not be too sad. Blame yourself.
First of all, the main function of platelets is to stop bleeding and coagulation. Then the question is, why does the increase in platelets cause bleeding symptoms? This time I will answer for everyone. Thrombocytosis is the mechanism of bleeding, which is mainly due to the number of platelets Increased, but there are defects in its functions, such as decreased platelet adhesion and aggregation, abnormal release function, decreased platelet third factor, and decreased 5-hydroxytryptamine. Some patients also have abnormal blood coagulation function, such as the decrease of fibrinogen, prothrombin, factor V, factor Ⅷ, which may be caused by excessive consumption of coagulation. At the same time, since most of the disease occurs in elderly patients, it may be combined with vascular degenerative changes, which may easily form thrombus, causing distal vascular infarction and rupture of the infarct area. In addition, due to too many platelets, activated platelets produce thromboxane, which causes a strong aggregation and release reaction of platelets, which can easily form microvascular embolism and further develop into thrombus. Primary thrombocythemia occurs in patients 50-70 years old, and the average age of onset is 60 years old. At the beginning of the onset, there are generally no obvious symptoms, and the main clinical manifestations are bleeding and thrombosis. Spontaneous bleeding is mostly bleeding from the nose, gums, and gastrointestinal mucosa; thrombosis is more common in foreign reports, but rarely in China. About 30% of patients present with functional or vasomotor symptoms, such as vascular headache, dizziness, blurred vision, burning pain in the palms and soles, peripheral numbness, and cyanosis. It can also show non-specific symptoms such as fatigue, fatigue, and insomnia. About 40% of patients may have hepatomegaly, and 80% of patients may have mild or moderate spleen enlargement. There are also about 20% of patients with this disease may have asymptomatic splenic infarction, which leads to spleen atrophy. Patients with thrombocytosis generally have a natural course, that is, the survival period, which is 7-15 years. However, if effective treatment is taken, if the platelets cannot be controlled in time and the platelets are too high, they will live at any time At the same time, some patients are prone to develop leukemia. Therefore, when the examination finds that the platelets are high, the treatment should be done in time to avoid delaying the best treatment time.
Glossopharyngeal neuralgia is manifested in paroxysmal severe pain in the subregion of the glossopharyngeal nerve. The nature of the pain is very similar to that of trigeminal neuralgia. It originates from the medulla oblongata, exits the skull from the jugular foramen together with the vagus nerve and accessory nerve, and is distributed in the tongue and pharynx. It is an important painful afferent nerve in the tongue and pharynx. Pain occurs on one side of the tongue, throat, tonsils, ear root and back of the mandible. Sometimes the main manifestation is ear root pain. It occurs between 35 and 50 years old. Whenever swallowing, chewing, yawning, and coughing It can induce pain and make people sleep and eat! Common symptoms of glossopharyngeal neuralgia 1 pain nature: paroxysmal severe pain, such as knife cut, jab, and painful convulsions. 2 Pain time: Frequent in the morning and in the morning, and attacks may occur during sleep. 3 Foreign body sensation and infarction sensation: There is foreign body sensation and infarction sensation in the pharynx and larynx at the onset of the disease, resulting in frequent coughing. 4 Pain triggers: Palpation can cause pain, also known as “trigger point”, which is common in the tonsil area, external auditory canal, and the base of the tongue. 5 The onset of trigeminal neuralgia is mostly intermittent, ranging from a few seconds to a few minutes. 6-severe patients may have arrhythmia, cardiac arrest, fainting, convulsions, seizures, laryngospasm, parotid hypersecretion, etc. Glossopharyngeal neuralgia is relatively rare. Patients with the disease will feel a sense of foreign body and infarction in the throat, which can cause tension in the patient. However, it is necessary to carefully check whether there is a foreign body in the oropharynx, and promptly treat the symptoms and recover as soon as possible!
After seeing a head CT result showing lacunar infarction, many patients will have doubts, is this a cerebral infarction? But I don’t have any symptoms. In clinical practice, many patients do not understand this problem. But to understand this problem is of great significance for many patients with lacunar infarction to develop cerebral infarction, so today I will introduce the similarities and differences between cerebral infarction and lacunar infarction. The difference between cerebral infarction and luminal infarction 1. Different infarcted blood vessels. The blood vessels of luminal infarction are mainly in the branches of brain tissue and deep perforating arteries. Because they are not the main blood vessels of brain tissue, they only cause small softening of the deep brain tissue in most cases. Focus, so the condition is also mild. However, cerebral infarction is much more serious. The diseased blood vessels of cerebral infarction may even occur in aorta or large vein, so the area of cerebral parenchymal necrosis will be larger. 2. The difference of lesion sites The most common sites of luminal infarction are mainly in the base of the pontine, internal capsule, basal ganglia, etc. However, because of the embolization of large and medium blood vessels in cerebral infarction, lesions can occur in the parts innervated by these blood vessels. 3. Different clinical manifestations. Because the infarcts are small, the infarcts usually do not have any symptoms, and it is difficult for patients to actively discover them. However, the onset of cerebral infarction usually has obvious symptoms, including weakness or numbness on one side of the body; numbness or skewed corners of the face on one side; slurred speech or difficulty in understanding language; eyes staring to one side; loss or blurred vision in one or both eyes; Dizziness with vomiting; disturbance of consciousness or convulsions, etc. 4. The size of the infarct size is different. The infarct size of the luminal infarction is small, usually less than 20mm in diameter, and it is more common in 5-15mm. The infarct size of cerebral infarction is usually more than 20mm or even larger. If not treated in time, there will be serious sequelae . 5. Different treatment methods For the treatment of lacunar infarction, its purpose is to prevent recurrence and prevent further occurrence of larger infarcts. Therefore, the treatment is mainly to correct high-risk factors and improve cerebral vascular circulation. The treatment of cerebral infarction requires every second. If the patient’s situation permits, thrombolytic therapy is best. This can minimize the patient’s sequelae and ensure the patient’s quality of life. The similarities between cerebral infarction and lacunar infarction are the same, that is, the high-risk factors that cause the disease are the same. Most studies have shown that advanced age, hypertension, diabetes, hyperlipidemia, atrial fibrillation, smoking, etc. are all high-risk factors that cause these two diseases. Therefore, to prevent the occurrence of these two diseases, it is necessary to effectively control blood pressure. , Blood sugar and blood lipids, and active treatment if you have heart disease. Finally, it should be emphasized that even asymptomatic lacunar infarctions should not be taken lightly, because patients who have experienced lacunar infarctions will have a high probability of developing cerebral infarctions if they do not pay attention to them. Seriously speaking, this is equivalent to burying in the brain. A time bomb, so we need to actively treat the cause. (Some of the pictures in the article originate from the network and the copyright belongs to the original author. I would like to express my gratitude to the author of the picture. If you find any infringement of your copyright, please contact me and I will delete it.)
How to care for the vas deferens after blocked vas deferens, postoperative, blockage, nursing Summary: Many people pay special attention to the treatment of vas deferens infarction. Experts say that there are still many options for clinical treatment of vas deferens, but there are still more options for surgical treatment. Then, do you know how to carry out nursing work after vas deferens infarction operation? Next, let’s take a look. Many people pay special attention to the treatment of vas deferens infarction. Experts say that there are still many options for clinical treatment of vas deferens, but there are still more options for surgical treatment. How should nursing work be done after vas deferens infarction operation? How to care after vas deferens infarction 1. Obey the doctor’s instructions and inspect for 2 hours after the operation. The general inspection for sticking or plugging is about 1 hour. For ligation, the inspection is about 2 hours. You can leave only when there is no swelling or bleeding in the operation area. 2. After going back, take antibiotics as prescribed by the doctor and make follow-up after operation. Discovery of blood in the wound, swelling, pain, swelling or blood stasis, and fever are all abnormal conditions and should be treated promptly. Summary of mild wound pain: Many people pay special attention to the treatment of vas deferens infarction. How to take care after vas deferens infarction 1. Obey the doctor’s advice and inspect for 2 hours after the operation. The general inspection for sticking or plugging is about 1 hour. For ligation, the inspection is about 2 hours. You can leave only when there is no swelling or bleeding in the operation area. 2. After going back, take antibiotics as prescribed by the doctor and make follow-up after operation. 1 Once blood is found in the wound, the swelling, pain, swelling or blood stasis, and fever are abnormal conditions and should be treated in time. Mild pain in the wound is normal. 3. If the seminal vesicle is not perfused during the operation, the sperm may remain for 4-6 months after the operation. Therefore, contraception should be adhered to. The semen can be monitored every 1 to 2 months until the absence of sperm is confirmed. Stop contraception. Even if the seminal vesicles have been perfused, the semen should be monitored after the operation, and contraception can be stopped only when there is no motile sperm. 4. Rest for 1 week after operation, avoid having sex within half a month, avoid heavy physical labor and strenuous exercise, such as cycling, playing ball, running, carrying burdens, etc. 5. When you resume your sexual life after the operation, you may feel pain and discomfort in the part of the operation. This is a common situation, not abnormal, and there should be no heavy mental burden. One month after the operation, if the local pain or swelling still persists, the patient should be treated for timely treatment. The above is the relevant introduction of how to care for the vas deferens after the vas deferens surgery. After reading the information, have you mastered it? Correct care is really necessary, because it is not only related to the patient’s disease as soon as possible Rehabilitation will also reduce the torture suffered by patients.
The clinical manifestations of polycythemia vera are usually bleeding tendency and thromboembolism, and the first cause of death in true red is thromboembolic complications. Among them, thrombosis is more common in middle-aged and elderly patients, and there are many treatment methods for thrombosis. Shi Shurong/Xie Bielu Director WeChat consultation platform zkxk9999 ☆ Anticoagulant drugs: such as heparin, argatroban, etc., inhibit the formation of infarction by inhibiting thrombin; ☆ antiplatelet agglutination drugs: such as aspirin, etc., can effectively inhibit platelet aggregation , Reduce thrombosis and prevent infarction; ☆ thrombolytic drugs: such as urokinase, plasminogen activator, etc. can also inhibit thrombosis, but may cause bleeding; ☆ improve microcirculation drugs: such as butyl benzene, human Urinary kininogenase, etc., can improve cerebral arterial circulation; ☆Chinese medicine treatment: use the method of promoting blood circulation to remove blood stasis to improve the blood environment, thereby preventing infarction ☆Surgery treatment: For patients with serious conditions, surgical treatment should be considered. The above is true red Patients with cerebral infarction should not buy medicines for treatment by themselves. The specific treatment should be communicated with the doctor before making a decision to avoid accidents. For more patient communication help, please follow the WeChat public account [True Red Patient Club] zkxy120
The surgeon tells you what should I do if I have not dared to eat for many years? Glossopharyngeal neuralgia is manifested in paroxysmal pain in the subregion of the glossopharyngeal nerve. The nature of the pain is very similar to that of trigeminal neuralgia. It originates from the medulla oblongata, exits the skull from the jugular foramen together with the vagus nerve and accessory nerve, and is distributed in the tongue and pharynx. It is an important painful afferent nerve in the tongue and pharynx. Pain occurs on one side of the tongue, throat, tonsils, ear root and the back of the mandible. Sometimes it is mainly manifested by ear root pain. It is common between 35 and 50 years old. Whenever swallowing, chewing, yawning and coughing Can induce pain, make people sleep and eat! Common symptoms of glossopharyngeal neuralgia 1 pain nature: paroxysmal severe pain, such as knife cut, jab, painful convulsions. 2 Pain time: Frequent in the morning and morning, and may have episodes during sleep. 3 Foreign body sensation and infarction sensation: Foreign body sensation and infarction sensation in the pharynx and larynx at the onset of the disease cause frequent coughing. 4 Pain triggers: Palpation can cause pain, also known as “trigger point”, which is commonly found in the tonsil area, external auditory meatus, and tongue base. 5 The onset of trigeminal neuralgia is mostly intermittent, ranging from a few seconds to a few minutes. 6-severe patients may have arrhythmia, cardiac arrest, fainting, convulsions, seizures, laryngospasm, excessive parotid secretion, etc. Glossopharyngeal neuralgia is relatively rare. Patients with the disease will feel a sense of foreign body and infarction in the throat, which can cause tension in the patient. However, it is necessary to carefully check whether there is any foreign body in the oropharynx, and promptly treat symptomatically to recover soon!
Damp and cold weather has become an important factor inducing cardiovascular disease. According to statistics, both the number of outpatients with acute myocardial infarction admitted to the emergency department and the number of outpatients have nearly doubled compared with summer. Experts remind that elderly people in winter need to pay close attention to changes in blood pressure, and heart disease patients should be reviewed regularly. Regular examination of electrocardiogram and cardiac vector diagram Electrocardiogram has progressive and characteristic changes, which is very helpful to diagnose and estimate the location, scope and evolution of the disease. Therefore, people with high risk of heart disease and myocardial infarction need to have regular medical examinations and check ECG. There are three types of ECG waveform changes in patients with myocardial infarction: 1. Waveforms in the necrotic zone: leads to the necrotic myocardium, with deep and wide Q waves. 2. The waveform of the damaged area: facing the lead around the necrotic area, showing the elevated ST segment. 3. Waveform in the ischemic area: Leads facing the periphery of the injury area show that the T wave is inverted. The typical ECG evolution process is: abnormal Q wave and ST segment are obviously elevated in the lead facing the infarct area at the time of onset (acute phase), the latter has a one-way curve with the T wave connected upward, and the R wave decreases or disappears ; Leads facing away from the infarct area show increased R wave and ST segment depression. Within days to 2 weeks after the onset (sub-acute phase), the ST segment gradually returns to the baseline level in the lead in the infarct area, and the T wave becomes flat or significantly inverted; the T wave in the lead facing the infarct area increases . From several weeks to several months after the onset (chronic phase), the T wave can be inverted in a V shape, with symmetrical limbs and sharp troughs. Anomalous Q waves often persist forever and T waves may recover within months to years. According to clinical data in my country, myocardial infarction located in the inferior wall, anterior septum, and localized anterior wall is the most common. Angina pectoris and myocardial infarction: the pain is similar but actually different. Angina pectoris is a clinical syndrome caused by insufficient blood supply to the coronary arteries, rapid myocardial ischemia and hypoxia. The pain is mainly located in the back of the sternum, and is more common in men. Most patients are over 40 years old. Fatigue, emotional excitement, satiety, cold, rainy weather, and acute circulatory failure are common causes. The pain nature and location of myocardial infarction are similar to those of angina, so patients are easy to confuse these two diseases. Frequency of attacks: Angina attacks are more frequent, and the duration of each attack is shorter, generally not more than 15 minutes; the pain of acute myocardial infarction is more severe and can last for several hours. Symptoms: Acute myocardial infarction is often accompanied by shock, arrhythmia and heart failure, and there is fever, which can not be relieved with nitroglycerin; angina pectoris is not accompanied by fever symptoms, and arrhythmia, shock and heart failure rarely occur. Examination: Angina pectoris rarely shows increased white blood cells, increased erythrocyte sedimentation rate, or increased serum myocardial enzymes. There is no change in the electrocardiogram, or there is temporary depression or elevation of the ST segment; while the electrocardiogram of acute myocardial infarction faces the infarct site The ST segment of the lead is elevated and there is an abnormal Q wave. The white blood cell count, creatine phosphokinase, aspartate aminotransferase, lactate dehydrogenase, myoglobin, and myosin light chain increased, and the erythrocyte sedimentation rate increased. Patients with heart disease can go to the hospital to find out the cause of pain or morbidity. The department of treatment is cardiovascular medicine.
The most direct consultation due to the problem of the epidemic, more and more people came online for consultation. I have often received such direct consultations recently: Doctor, can the cerebral infarction return to normal? As a doctor, how to answer this rich question? In general, I want to ask if there is a film, the basic condition of the patient, how long it has been onset, what are the symptoms, and how to treat it. After these questions are clarified, we still need to ask the patient if there are any basic diseases, how to control them, and how effective is the control? That’s it, or some patients are impatient, and ask me: can you say okay? Doctor’s explanation When a patient sees a doctor, the doctor must ask the patient’s basic information, including age, height, weight, when the symptoms appear, how long it has been, whether there are other diseases, what medicines are used, and the How about waiting. Afterwards, check the body, including looking at and touching. It also depends on all the examinations and tests of the patient, and further examinations are possible. If the patient is not the first visit, he still has to ask how to treat it, the changes after treatment, etc. On this basis, the prognosis of the patient is judged, and even the accurate prognosis cannot be completely judged, and it is necessary to judge while treating. But online consultation, if you ask like this, the patient has long been impatient, generally I first look at the patient thinks the key, such as film, symptoms, and then ask a little bit more information. After fully understanding the situation, make a judgment, possible prognosis, and give advice on treatment and daily management. What factors are involved in the prognosis of cerebral infarction? Cerebral infarction, the prognosis can not recover this question can not be generalized. Because cerebral infarction refers to the infarction of functional cerebral blood vessels, brain function is damaged. Symptoms vary depending on the supply vessel and the size of the vessel. 1. It is related to the size of cerebral infarction. For example, small blood vessels are blocked, the cerebral ischemic range is small, collateral circulation is easy to form, recovery is faster, and the prognosis is better. For example, the blocked blood vessels are large, the cerebral ischemic range is large, the brain tissue is seriously damaged, the clinical symptoms recover slowly, and the prognosis is poor. 2. Cerebral infarctions of the same size related to the location of cerebral infarctions occur in different locations, with different symptoms and greater prognosis. For example, if it occurs in the brainstem, the symptoms will be severe or even coma, while in the cerebellum, it may have little effect. 3. After the occurrence of cerebral infarction, it is generally related to whether the treatment is timely. Seek medical treatment within 4-6 hours. You can intervene for thrombectomy or thrombolysis. If you can recanalize, you may completely recover, but the current recanalization rate is less than half. There are also post-treatments to prevent the infarction from expanding further, and later rehabilitation exercises are directly related to the prognosis. 4. The first attack is related to the number and number of infarctions, and the prognosis is good. But a large area of infarction has a poor prognosis. More than two infarctions, especially on both sides of the cerebral blood vessels involved, have a poor prognosis. The more infarcts, the worse the prognosis. Those with a single infarct have a better prognosis. 5. It is related to the nature of emboli, such as loose emboli. During the operation with blood, it breaks itself up and flows to the far end of the blood flow. If it blocks small blood vessels, the prognosis is better. Fat emboli, air emboli, and bacterial emboli have a worse prognosis than cardiogenic emboli. However, those with cardiogenic emboli who cause brain abscesses have a poor prognosis. 6. It is related to whether there are complications such as bedsores, lung infection, urinary tract infection, diabetes, coronary heart disease, arrhythmia, heart failure, etc. The prognosis is poor, and those without complications have a better prognosis. 7. The age is related to the patient’s age, poor constitution, and poor prognosis. Young age, good health and good prognosis. However, a large area of cerebral infarction, or brain stem infarction, the patient will have serious symptoms from the onset of symptoms, or even coma. If admitted to the hospital within 6 hours, through interventional thrombolysis, or intravenous thrombolysis, if it passes, most of them Can return to normal. But after a long time, there may be sequelae. But for those cerebral infarctions that are not recanalized, the patient’s sequelae must be present and will not return to normal. But through the later exercise and functional recovery, some functions can be restored to some extent, and even some cannot be recovered at all. Therefore, to determine whether the cerebral infarction can recover, it is related to too many factors. During the consultation, the information and materials must be prepared, so that the doctor can judge relatively accurately and give specific opinions, otherwise it can only give Suggestions in principle.
Stroke is a diagnosis of Chinese medicine. Western medicine is also called stroke. It is divided into cerebral hemorrhage and cerebral infarction. The former is a blood vessel rupture and the latter is a cerebral blood vessel blockage. Stroke can cause many sequelaes according to the severity, such as limb paralysis, language disorders, paresthesia, and memory loss. Lameness is a type of limb paralysis caused by stroke. From the cause, it is definitely a brain problem. Brain Structure and Dominating Functions The human brain is located in the head and consists of six parts: the brain, cerebellum, diencephalon, midbrain, pons, and medulla oblongata. The brain is the most complex part. It controls most of the body’s functions and activities. The brain has left and right cerebral hemispheres, which are structurally distinguishable and can be divided into frontal lobe, parietal lobe, temporal lobe, occipital lobe, marginal lobe, internal capsule, and basal ganglia. From the innervation function, they can be divided into three parts: brain The nucleus, limbic system and cerebral cortex. The brain nucleus mainly controls the basic life activities of the human body, such as breathing, heartbeat, exercise, sleep, balance, and sensation, while the limbic system is responsible for action, emotion, and memory, as well as the regulation of body temperature, blood pressure, blood sugar, etc. The cerebral cortex is responsible Is more advanced cognitive and emotional functions. What is the problem with stroke and lameness? Stroke is caused by ischemia and hypoxia caused by cerebral vascular embolism. The arteries supplying the brain can be divided into anterior and posterior circulation. The anterior circulation mainly includes internal carotid artery, middle cerebral artery, anterior cerebral artery, anterior choroidal artery, and posterior circulation. Vertebral basilar artery and posterior cerebral artery. In general, hemiplegia caused by infarction of the internal carotid artery, anterior cerebral artery and middle artery in the anterior circulation is more common. If the internal carotid artery is occluded, if the collateral circulation is poor, it can cause complete contralateral hemiplegia in severe cases. Hemiplegia on the lower limb is heavier than the upper limb. Later circulation is more common in vertebrobasilar infarction, which can cause infarcts at the junction of brain stem, cerebellum, thalamus, occipital lobe and temporal parietal occipital junction, leading to sudden vertigo, vomiting, facial or quadriplegic paralysis. As far as cerebral infarctions are concerned, hemiplegia often occurs in frontal lobe, internal capsule and basal ganglia. Therefore, if you want to locate the infarcted part of the brain, you can use skull CT or magnetic resonance scanning. If you want to find an embolism in that blood vessel for thrombectomy or thrombolysis, cerebral angiography can be performed. When cerebral infarction occurs, time should be the first factor in determining the prognosis of the patient. The sooner the effective intervention, the fewer the number of brain cells will be necrosis due to ischemia and hypoxia, and the faster the patient will recover, with limb paralysis or dysfunction The lower the chance of being effective. This is a special reminder that if you notice weakness on one side, slurred speech, numbness on one side, or asymmetry, you must immediately call 120 to the hospital for emergency treatment. If it is a cerebral infarction, time is the brain, It can avoid the sequelae of leg lameness after stroke. (Some of the pictures in the article are from the source network. The copyright belongs to the original author. Thank you for the picture author. If you find any violation of your copyright, please contact me and I will delete it.)
Being diagnosed with thrombocytosis, the morbid performance has these two points! The first point: the disease is a clonal disease of pluripotent stem cells, mainly showing the increase in the number of platelets; the second point: in addition to the problem of platelet increase, its function is also defective, such as platelet adhesion and aggregation function decline, abnormal release function , Decreased platelet third factor, reduced serotonin, etc. Some patients also have abnormal blood coagulation functions, such as the reduction of fibrinogen, prothrombin, factor V, factor VIII, which may be caused by excessive consumption of blood coagulation. Thrombocytosis needs attention-thrombosis! Because most of the disease occurs in elderly patients, it may be associated with degenerative changes of blood vessels, and it is easy to form thrombosis, causing infarction of the distal blood vessels and bleeding in the infarcted area. In addition, due to excessive platelets, activated platelets produce thromboxane, causing a strong aggregation and release reaction of platelets, easy to form microvascular embolism, and further develop into thrombosis. Remarks: Probability of clinical manifestations of thrombocytosis 30% of patients present with functional or vasomotor symptoms at the time of treatment, such as vascular headache, dizziness, blurred vision, burning sensation in palms and soles, peripheral numbness Cyanosis etc. It can also be manifested as non-specific symptoms such as fatigue, fatigue, and insomnia. 40% of patients may have hepatomegaly; 80% of patients may have mild or moderate splenomegaly. In addition, about 20% of patients with this disease may have asymptomatic splenic infarction, which causes spleen atrophy. If you have any questions about this article or your condition, follow the patient’s WeChat account to get help: xejb120