. . . First of all, it is stated that some of the treatments in this article are empirical, only some personal thoughts, and I am not a big coffee, so it cannot be used as an academic authority and guidance, only for the discussion of patients and peers. , Wrong, please criticize and correct me.  . . . . . The writing of this article is also based on frequent consultations with such questions, not only in the clinic but also online, and often consulted on how my recurrent candida (mould) vaginitis treatment.  . . . . . Vaginitis, for every woman, may be a clinically very common disease that is unavoidably tormented by her life. Candida vaginitis is a kind of vaginitis that may make some women feel headache. Because of its repeated attacks, people are more tortured.  . . . . Candida vaginitis is also known as fungal vaginitis, but it is now more scientifically named vulvovaginal candidiasis (abbreviated as VVC), and its incidence is second only to Bacterial vaginosis. 80% to 90% of the pathogens are Candida albicans, which are opportunistic pathogens. Common causes include pregnancy and diabetes, and large amounts of immunosuppressants and broad-spectrum antibiotics.  . . . . The most common symptoms of candidal vaginitis are leucorrhea, burning and itching of the vulva and vagina. Among them, the main reason for visiting a doctor is itching, because itching in a particular part brings a very bad feeling to the patient and inconvenience in socializing. Its typical leucorrhea is curd-like or okara-like, and many experienced people can make a preliminary diagnosis based on this.  . . . The treatment is relatively simple, and there are many drugs to choose from. I remember when I first went to work, Dakning and Baofukang suppository were actively promoted clinically as drugs for the treatment of vaginitis. Dakronin, in particular, emphasizes its unique clinical value due to different dosage forms, including the treatment of recurrent mycotic vaginitis.  . . . . The treatment of vulvovaginal candidiasis is relatively simple, often through the application of drugs such as Dakron, nystatin vaginal suppository, terbinafine vaginal effervescent tablets, and even some of our Cheng Yao, such as Baofukang suppository and Kushen gel, can all play a very good therapeutic role. Most people get rid of the medicine once they have taken it. If they relapse occasionally, they can do it again. However, there is a group of people who often attack and disturb their lives. This type of vulvovaginal candidiasis is called recurrent vulvovaginal candidiasis (abbreviated as RVVC).  . . . .RVVC is defined as having at least 3 symptomatic episodes within 12 months, but some researchers claim that an additional episode is required, in other words, 4 episodes.  . . . . Why are there frequent attacks?  . . . .Some studies believe that long-term oral contraceptives, pregnancy, combined with other vaginal diseases, history of artificial abortion, long-term application of immunosuppressive agents, long-term use of pads, tights, and use of intrauterine devices are RVVC. The related risk factors are also related to combined diabetes and long-term use of broad-spectrum antibiotics, estrogen and corticosteroids. Another cause is obesity, which is also a cause of RVVC.  . . . . Frankly speaking, at present, I think that the cure of RVVC is still a problem. I hope that a certain method or a certain doctor can completely cure RVVC and never relapse. That is still a distant one. Dream, although this is just a small inflammation. Therefore, the focus of our current treatment is to actively find and remove the incentives to prevent relapse.  . . . . For the treatment of RVVC in patients with diabetes, controlling blood sugar has become an important part. Based on other incentives
Because syncope is episodic, and some symptoms are similar to epilepsy, such as loss of consciousness, dilated pupils, and even convulsions, incontinence, etc., it is easy to be misdiagnosed as epilepsy. Most syncopes have obvious incentives, but epilepsy has no obvious incentives and thus suddenly attacks. Most syncope occurs in the upright position, partly in the sitting position, rarely in the lying position, and epilepsy is not necessarily. Syncope attacks generally fall slowly, while epilepsy falls suddenly. The syncope is mainly slow waves, and the EEG is normal after the syncope, and the epilepsy usually has corresponding discharge waves. Before syncope, there are often prodromal symptoms such as dizziness, vertigo, paleness, abdominal discomfort, etc., and less epilepsy in children. The difference between syncope and epileptic seizures: . The face of the syncope is pale, blood pressure is reduced, and the pulse is slow and weak. However, his face became cyanotic during his major attack, his blood pressure (blood pressure food) was not low, and his pulse increased. Tongue biting and urinary incontinence are rarely seen in syncope but more often in epileptic seizures. The convulsions caused by syncope are rare, and even if they are, they last for a very short time, usually within 15 seconds, while the time of a major seizure is relatively long. There is apnea during a major attack and generally not during syncope. Major seizures often have post-onset drowsiness and positive neurological signs, while syncope generally does not.
Thrombocytosis is more common in middle-aged and older people, but in clinical hematology, many children have thrombocytosis. Then, according to the different causes and performances of thrombocytosis, their harm to the human body is also different. The following will explain one by one: Director Shi Shurong’s micro-signal zkxk99991. What is the standard for increasing the number of platelets? Note: When the automatic blood cell analyzer prompts an increase in the number of platelets, peripheral smear microscopy should be further performed. The purpose is to prevent the instrument from misrecognizing broken red blood cell fragments as platelets, except for pseudoplatelet increase; at the same time, the platelet morphology can be observed as the etiological diagnosis. Provide clues. 2. What is thrombocytosis? Divided into primary thrombocytosis (Essentialthrombocytosis, ET) and reactive thrombocytosis (Reactivethrombocytosis, RT). (1) Primary thrombocythemia is a myeloproliferative disease. It is rare in pediatrics due to abnormal single or polyclonal hematopoietic stem cells or biological abnormalities of TPO. (2) Reactive thrombocytosis is caused by various blood or non-blood diseases stimulating the generation of megakaryocytes, and is a common secondary thrombocytosis in pediatric clinic. 3. What are the common causes of reactive thrombocytosis? Reactive thrombocythemia refers to an increase in platelet counts caused by internal and surgical diseases other than hematological diseases in the absence of hematological diseases such as myeloproliferative or myeloproliferative disorders. Common causes include: acute infections, inflammatory reactions, malignant tumors, etc. After these conditions have resolved, the platelet count will return to normal. What are the common conditions that may cause reactive thrombocytosis? (1) recent trauma; (2) recent surgery; (3) previous surgery to remove the spleen; (4) recent fever, infection, inflammation, etc.; (5) recent loss of weight, sweating, fatigue, etc. Performance, suggesting that there may be malignant tumors! 4. What are the diagnostic criteria for thrombocytosis? The complications of thrombocytosis are mainly thrombosis and hemorrhage. It usually occurs when there is a significant increase in platelets. The chance of hemorrhage of primary thrombocytosis is much higher than the incidence of thrombosis. The clinical manifestations of secondary thrombocytosis are mostly benign. The above complications are rare. Report. The diagnostic criteria for primary thrombocythemia are as follows, which can be diagnosed by satisfying the first 3 or Articles 1 and 3-5: (1) The platelet lasts more than 450×109L; (2) The presence of pathogenic mutant genes (such as JAK2 or MPLTpo receptor mutation); (3) Excluding other myeloproliferative diseases (especially CML, MDS, true red, or myelofibrosis); ⑷ No incentive for thrombocytosis and normal iron storage; ⑸ Increased bone marrow megakaryocytes, abnormal morphology, reticular fibers usually do not increase . 5. How to treat thrombocytosis? Among them, secondary thrombocytosis is mainly based on the treatment of primary disease. When the platelets are 1000×109L and there are risk factors, low-dose aspirin, dipyridamole and other anti-blood can be given… For the treatment, hydroxyurea and interferon are conventional drugs, but because of the inevitable side effects, long-term application is not recommended. At the same time, patients can cooperate with targeted TCM syndrome differentiation to increase efficacy and reduce toxicity! For more thrombocytosis disease knowledge or patient help, you can pay attention to WeChat public number: xxbzd999
The small bubbles that appear on the hand may be sweat herpes. Sweat herpes is generally more likely to occur in the particularly developed parts of the sweat glands, such as hands and feet, which can be manifested as blisters on the hands and feet. It is thought to be caused by sweating and other factors, so it is called sweat herpes. However, it has been confirmed that the occurrence of sweat herpes is not directly related to sweat glands and sweating. Its clinical manifestations are deep vesicles, millet to rice grain size, slightly higher than the skin surface, often without blush. Symmetry occurs on the palmar plantar and finger (toe) side. After 1-2 weeks, it dries up and turns into swarf, which can occur repeatedly, with varying degrees of burning and itching, often for several consecutive years. At present, the etiology of sweat herpes is still not very clear in medicine. Only a few patients find that it is related to fungal infections or metal allergies and even emotional stress elsewhere. May be related to mental stress, sweating in hands and feet, fungal infections and allergies, which are more common in summer. The treatment of sweat herpes is divided into two parts: ① The treatment of the symptoms and damage of sweat herpes is divided into two ways: local topical and systemic (systemic) medication. The topical topical medications mainly focus on convergence and antipruritic. Agents, antihistamines, oryzanol, etc.; ② For the treatment of the possible causes or inducements of sweat herpes, inquire about the history of sweat herpes, find out the possible causes and remove them (for example: nickel allergy should be low nickel diet) .  .
Because syncope is episodic, and some symptoms are similar to epilepsy, such as loss of consciousness, dilated pupils, and even convulsions, incontinence, etc., it is easy to be misdiagnosed as epilepsy. Most syncopes have obvious incentives, but epilepsy has no obvious incentives and thus suddenly attacks. Most syncope occurs in the upright position, some occurs in the sitting position, rarely occurs in the lying position, and epilepsy is not necessarily. Syncope attacks generally fall slowly, while epilepsy falls suddenly. The syncope is mainly slow waves, and the EEG is normal after the syncope, and the epilepsy usually has corresponding discharge waves. Before syncope, there are often prodromal symptoms such as dizziness, vertigo, paleness, abdominal discomfort, etc., and less epilepsy in children. The difference between syncope and epileptic seizures: . The face of the syncope is pale, the blood pressure decreases, and the pulse is slow and weak. However, his face became cyanotic during his major attack, his blood pressure (blood pressure food) was not low, and his pulse increased. Tongue biting and urinary incontinence are rarely seen in syncope but more often in epileptic seizures. The convulsions caused by syncope are rare, and even if they are, they last for a very short time, usually within 15 seconds, while the time of a major seizure is relatively long. There is apnea during a major attack and generally not during syncope. Major seizures often have post-onset drowsiness and positive neurological signs, while syncope generally does not.
& nbsp. & nbsp.Statistical data show that the child suddenly fainted. If environmental factors are excluded, the rest is the cause of epilepsy. The age of onset is mostly 7-15 years old, and more than half of them are concentrated in 12-15 years old. With the increase in the use of electronic products, there is a trend that children’s epilepsy may have signs of early advancement. & nbsp. & nbsp. → _ → Several practical suggestions of famous epilepsy doctors, click here to ask & nbsp. & nbsp. Parents should note that if the child has long pain, cramps, stunting, irritability, anorexia, limb paralysis, convulsions and other manifestations To be alert, first see if it is caused by calcium deficiency. If not, you must pay attention to the cause of epilepsy. & nbsp. & nbsp. Take children to outdoor activities, run and jump, play basketball and other sports and sunbathe outdoors. The chance of spring epilepsy is significantly higher each year than in other seasons. Especially in May, it is called “the magical May of epilepsy”. & nbsp. & nbsp. There are many types of triggers that cause seizures. Most people suffering from epilepsy are more concerned about the cause of the disease and do not know much about the origin of the disease. Recommended reading: What is the cause of epilepsy caused by children twitching and rolling their eyes? First of all, epilepsy patients are devoted to clinical observation, caring about their epilepsy every time, find out which line of patients they belong to, and encourage Be firm in yourself.
Recently, I have received a lot of private messages from my friends. I basically asked if there are any signs of hair loss in advance. The hair loss was already serious when I discovered it. My partner, in fact, in all types of hair loss, except for the alopecia areata that we said happened suddenly, other hair loss has a symptom, such as from a little drop, to a drop, the hair loss will be in the process. These are warnings to you. The following four “signals” that are aggravated are the most common. Four common hair loss signals 1. Sudden increase in hair loss. Everyone must know that hair loss is below 100 hairs per day, which is normal. If one day you shampoo or wake up, the hair loss on the pillow increases, and the number of hair loss per day for more than 100 days is the first sign of hair loss. At this time, if you find out in time, you must adjust your life laws and eating habits. At this stage, hair loss suddenly increases, mostly due to mental state and hormones. Do not stay up late, eat a light diet, do some outdoor sports in a timely manner, participate in group activities, relax your spirits, and hair loss can be well relieved. 2. Hair becomes thin and soft One day when you are sorting your hair, you suddenly look at the hair in your hand and it seems to become thinner and softer than before! This should be the case of blocked hair follicles. Blocked hair follicles affect the absorption of nutrients by the hair, slow down the growth rate of the hair, and make the original thick hair become thin and soft. Under normal circumstances, clogged hair follicles will not cause direct hair loss. Even for seborrheic hair loss, which is most closely related to oil secretion, the root cause of hair loss is also male hormones, not oil. Androgens stimulate the sebaceous glands to secrete too much oil, which leads to damaged hair follicles and hair loss. Oil and hair loss are side by side, not causal. 3. Increased greasy hair and dandruff. Sudden increase in hair oil production is often caused by endocrine disorders or excessively strong hormones. For male hair loss, the increase in oil production is when the amount of hair loss increases. The increase in dandruff is due to fungal infection of the scalp. If there is too much dandruff for a long time and it is difficult to remove it, it proves that the scalp environment has been destroyed, which may cause folliculitis. 4. Hair loss at the age of 20 Our hair growth was about 25 years old, but now some hair loss partners start losing hair at the age of 19 and 20. In this case, there is a high probability of genetic hair loss. If hair loss occurs between the ages of 30 and 40, the influence of genetics is not too obvious, or it is a good lifestyle. In this case, it is easier to control hair loss. & nbsp. & nbsp. How to avoid the hair loss inducement is still the same old saying, we can reasonably avoid these avoidable inducements of hair loss. For example, if you often stay up late, the body will secrete hormones to maintain the mental state of the person. If the hormone secretion increases, your endocrine will be imbalanced, increasing the risk of hair loss. In addition to staying up late, I often eat spicy and irritating foods, the diet is relatively greasy, and my spirits are often in a tense state. Frequent perm and hair coloring are common causes of hair loss. These friends must keep in mind in real time! Reducing physical damage mainly refers to not pulling hair in everyday life. For example, women should not tie ponytails that are too high and too tight, and often change the position of slits to avoid stubborn slits and so on. & nbsp. & nbsp. Timely treatment There are many small partners who think that hair loss will be good for a while, so do n’t worry about treatment. This idea is only suitable before the first signal is issued, if your hair loss problem is serious, you must take timely measures, not to avoid medical treatment. Therefore, the causes of hair loss should be completely solved. Even if it is male, the precious hair follicle resources must be protected. It is absolutely necessary to break the jar.
I believe that everyone knows more or less that cervical spondylosis is a relatively stubborn disease, and its treatment and rehabilitation require a long process. In addition, after treatment, many patients with cervical spondylosis may relapse, so how can the reason for cervical spondylosis relapse be explained by scientific medical science? & nbsp. & nbsp. One of the incentives: anatomical and physiological functions of the cervical spine cause recurrence. & nbsp. & nbsp.The cervical spine is a segment of the spine with a high degree of mobility and a weak structure. Compared with the thoracic and lumbar vertebrae, the cervical spine is more active; the cervical vertebral body, posterior joints and other structures are weaker than the thoracic and lumbar spine structures, and unlike other spine spine, there are thorax, abdominal muscles, hip Bone, etc. is used as an aid, so the stability of the cervical spine is poor. & nbsp. & nbsp. A large degree of activity and poor stability form a pair of obvious contradictions, whether it is muscle spasm caused by wind cold in the neck muscles, or cervical spine stability damage caused by sprains, sleeping positions or improper pillows, etc. May cause new dislocations and recurrence of symptoms. & nbsp. & nbsp. The second cause: recurrence of the structure around the cervical spine & nbsp. & nbsp. The structure around the cervical spine is more complicated, with important blood vessels (including the push artery passing through the transverse foramen that other vertebral bodies do not have), nerve tissue, plus The transverse foramen of the upper cervical spine is small, and it is often hyperplastic and obvious, which is also one of the factors that are prone to relapse. & nbsp. & nbsp. The third cause: fatigue and head and neck trauma leading to recurrence. & nbsp. & nbsp.As mentioned above, when suffering from cervical spondylosis, due to its poor stability, even after it is cured, its stability will take some time to go Restore and consolidate. During this period, many factors, such as sudden turning back, overwork, and neck injury caused by sudden parking while riding, are likely to cause the recurrence of cervical spondylosis and bring great harm to patients and friends. & nbsp. & nbsp.